Brain Research Institute, Niigata University Field for Evolutionary Brain Pathology Department of Neuroscience of Disease Care fish, Cure human
Godfried Dougnon

Godfried Dougnon

Associate Professor

Career

December 2013: Graduated from Abomey-Calavi University, School of Pharmacy (pharmacist license) April 2016: Research student, Graduate School of Pharmaceutical Sciences, Kyoto University (MEXT Fellowship, Embassy Recommendation) April 2017: Entered master's program, Kyoto University March 2019: M.Sc. (Pharmacy), Kyoto University April 2019: Entered doctoral program, Kyoto University March 2022: Ph.D. (Pharmacy), Kyoto University April 2022: Assistant Professor, Department of Neuroscience of Disease, Brain Research Institute, Niigata University September 2026: Associate Professor, Department of Neuroscience of Disease, Brain Research Institute, Niigata University

In their words

I was born and raised in the Republic of Benin (West Africa). I am a native French speaker, and growing up I came into connection with Japan through my favorite anime, Naruto. I see myself in this kid, who worked so hard into making his dream become a reality (smile). My passion for research was born since I was a child, and I always wanted to be a scientist, someone who can discover great things and make an impact to the world. This led me to becoming a Pharmacist and come to Japan to pursue my dream. Like the kid I mentioned before, I hope to use my passion for research to contribute to a better world, especially for neurodevelopmental disorders such as ADHD, ASD and other brain diseases (PD, AD).

Selected publications

  • Dougnon, G., Otsuka, T., Nakamura, Y., Sakai, A., Yamanaka, T., Matsui, N., Nakahara, A., Ito, A., Hatano, A., Matsumoto, M., Igarashi, H., Kakita, A., Ueno, M., Matsui, H.*. A protective role for APP in nuclear waste clearance via lysosomal exocytosis. Proc. Natl. Acad. Sci. USA. 123(24):e2524190123. 2026. https://doi.org/10.1073/pnas.2524190123
  • Dougnon, G., Matsui, H.*. Environmental context modulates sociability in ube3a zebrafish mutants via alterations in sensory pathways. Mol. Psychiatry. 31(1):158-172. 2025. https://doi.org/10.1038/s41380-025-03180-0